A Moral Issue Of Murder essays

A Moral Issue Of Murder essays Capital punishment is an issue that has long been debated amongst Americans. We have been questioning the morality of an Eye For an Eye way of thinking. Many say that serious crimes deserve serious punishments such as death, where others view death to be an unjust punishment for any crime. Each side seems to be equal in its volume of supporters. There are also many who ride the fence on the issue, unconvinced of either side. I, however, am writing to illustrate the immoral view of capital punishment. This document does not presume to have the right answer to the question, for to assume that would be arrogant. No, this essay is merely a tool to be used in weighing this very important issue. When the unspeakable happens and a human life, or lives, is taken by another, for reasons other than self defense or accidental, revenge and fury are so often the first emotions to surface. It is often thought that restitution will ease the pain of loss. To accomplish the feat to revenge wounded hearts, many turn, quite ironically, to murder. That is after all what capital punishment entails, murder. However justified, to intentionally end a life is to murder. At first glimpse, to take a life for a life taken seems with just cause and the right of society. It appears to be the only one fair solution. But is it okay for society to kill a killer? Is the right truly given to humans to deal out judgment and death? Should it be our decision who shall live and die? Or does only one truly posses the gift to decide? These questions make up the backbone of this controversy and need to be answered. I find it odd when Ernest Haag states, I see no evidence for society somehow not having the right to execute murderers. It has always done so. Traditional laws and Scriptures have always supported the death penalty(640). When the Eighth Amendment of the Constitution of the United States ...

What Really Is The Best Headline Length

What Really Is The Best Headline Length How long should my headline be? Youve asked yourself this, and with good reason. In the content marketing world, a lot of  attention has been given to the headline of your blog post in recent years. Theres so much headline information out there, so many dos and donts, so many great tips that youre drowning in advice and you have no idea of what to actually do. You want a definitive answer in the form of a number or at least a firm set of guidelines. You dont want to hear  it depends. The truth is, it does depend. Sort of. It depends on what you want your headline to do. Do you know what you want from your headline? What Really Is The Best #Headline Length? #blogging via @JulieNeidlinger Write The Best  Headlines With A  Free Headline Analyzer The headline analyzer  will help you: Use headline types that get the most traction for social shares, traffic, and search engine ranking. Make sure you have the right word balance to write readable headlines that command attention. See the best  word and character length for search engines like Google and email subject lines, while also seeing  how your readers will scan your headlines. Try The Free Headline Analyzer Now How To Write The Best Headlines The Old School Way There are two schools of thought when it comes to headlines. Traditionally, headlines should be, according to Jakob Nielsen: Short, typically 5 words or less. People dont read very much online. Information-rich. The headline should summarize the article. Starts with keywords. People tend to scan, and read the first words most of all. Understandable, even out of context. Headlines often appear on their own, without articles. Especially in social media and search engine results. Predictable and match reader expectations. Dont frustrate your reader. They should get what they expect when they click. Nielsens shorter headlines arent the label variety. Label headlines are those which are short, but not terribly information rich. For example, a Nielsen headline might be Speeding Car Crashes Into Wall while an uninformative label-type headline might read Speeding Car. I Want My Headline To Perform Well On Social Media The second school of thought, however, is what we tend to see online now. And it seems to be  the exact opposite of what Nielsen says. A headline that performs well on social media is one that people share a lot, and click through a fair amount. But mostly share. The headline is often shared as much as the people sharing it will read. This is a unique situation in headline history. Instead of being helpful in the traditional sense (as described above), headlines are now meant to  invoke curiosity and capture clicks. Write #headlines to invoke curiosity and capture clicks. via @JulieNeidlingerThis is particularly important on social media, where news feeds are quickly cycling through, competition with other headlines is fierce, and you have to pull out all stops to get someone to notice. Long, rambling, and sometimes bordering on bizarre. Headlines now are often quite long and almost a story unto themselves. Though that is shifting somewhat from 2014 (the year of Upworthy-styled headlines), headlines still maintain a longer approach than Nielsen might recommend. Emotion-rich. What the article is about doesnt matter. Emotion does. Keywords are sparse. Few keywords, with more attention given to emotional subtexts. Not much context. Without the article, the headline is a worm on a hook and little more. Rely on shock/emotion/curiosity factor. Readers dont know what to expect, and thats why they click. How does that speeding car headline read for the social media age? How about 5 Reasons You Should Fear That Speeding Car. The social messages that accompany your blog posts wont necessarily be your headline. Youll probably customize them for repeated shares. But if you do rely on your blog post headline to be the body of the social message, then the ideal social message length will matter for your headlines. Buffers Kevan Lee has written a seriously comprehensive blog post about the ideal lengths of just about anything you could put online. Its packed with information on the length of just about everything, but check out what he discovered for the most popular social networks: Twitter: 71–100 characters Facebook: 40 characters Google+: 60 characters LinkedIn:  80–120 characters Depending upon which network you lean most heavily or get the most traction, you should consider making your headline fit the ideal length for that social  network. Within those character ranges, remember the importance of emotions in a headline that does well on social media. Emotional headlines, with specific trigger words, get shared more on social media. I Want My Headline To Do Well In Email Just like social media, you have the ability to customize your emails so that you rework how your headlines appear in the subject lines. But not everyone does. Sometimes the work you put into finding that perfect  headline can carry right on through to your email subject line. So how long should they be? MailChimp, the popular email provider, has stated that it doesnt really matter how long your subject lines are. Last year, we carried out our own tests on how our email subject lines fared. We found that an email subject line with around 50 characters fared the best. The conclusion? Find out what works for you by testing if you dont believe either MailChimp or our results. As you test your headlines in your email to determine what works, remember the difference between open rates and click-through rates. Open rates are not the same as click-through rates. An open rate is the percentage of total recipients that opened the email. In some email software, just by opening their inbox they open emails. The click-through rate is the percentage of those who opened and then clicked at least once. Aim for a high click-through rate as the basis of what a successful headline does, not just an open rate.

Ethics in health Essay Example | Topics and Well Written Essays - 2250 words

Ethics in health - Essay Example The devastating impact of child maltreatment on individuals, families, and society at large is well documented in empirical and clinical studies (Gilbert, 1994). Many serious long-term effects have been linked to child maltreatment, including mental retardation, intellectual and intelligence handicaps, impaired aggressive impulse control, diminished ego competency, reduced reality testing, and poor interpersonal relationships. Child maltreatment results in increased antisocial activities. Maltreated aboriginal aboriginal children have more serious personal problems and engage in more antisocial activities and violence toward themselves and others (Hutchinson, Dattalo and Rodwell, 1994). When older, they end up in juvenile and adult correctional facilities at higher rates than aboriginal children from the general population. It is evident that child abuse and neglect is a problem that affects not only the individuals and families directly involved, but all sectors of society. Therefore, in order to deal with this problem, it is necessary for all professionals from all aspects of human ecology (individual, family, community, society, world) to become involved. Aboriginal children throughout the world suffer an array of threats to their development, well-being, and survival (Lindsey, 1994). They suffer from poverty, famine, disease, and war. They suffer as they navigate the child-rearing practices and rites of their diverse cultures. And, they suffer from acts of omission or commission by their individual parents and caretakers. Parental behavior that compromises the development and survival of their offspring seems to contradict the biological and cultural dictates of rearing the next generation (Pecora et al, 1995). This enigma of human behavior demands consideration from a wider range of human cultural adaptation than that afforded by Western societies alone. This chapter will consider definitional issues that have been an impediment to cross-cultural research on child maltreatment. It will then turn to a review of current knowledge concerning categories of aboriginal children vulnerable to abuse, the relationship of kinship and social networks to child maltreatment, and the impact of urbanization and social change. Child abuse is defined as any action (or lack of) which endangers or impairs a child's physical, psychological or emotional health and development. There are many factors that constitute child abuse (Pelton, 1989): Physical Abuse - is any physical injury to a child which is not accidental. This involves severe beating, shaking, burns, human bites, strangulation. Emotional Abuse - is when a child is not nurtured and is not provided with love and security. This involves constant criticism, belittling and persistent teasing. Sexual Abuse - is when the child is involved in any sexual activity with an adult. This involves fondling, exhibitionism, sexual intercourse, incest, pornography. Neglect - is depriving a child of their essential needs. These include nutrition, clothing, warmth and shelter, emotional security and protection, medical and dental care, hygiene, education and supervision. According to Campbell in 1999, a clinical doctor, every case of child abuse leads to permanent damage and great long-term suffering. It may also lead to psychological trauma. Caplan in 1994 defined psychological trauma as the unique individual experience of an event or enduring conditions, in which: 1.

Major Problems in Dealing with Health in Rural India and Advise on how Article

Major Problems in Dealing with Health in Rural India and Advise on how to Address the Problems - Article Example Even though the government has introduced various health policies for example the 2005 National Rural Health Mission (NRHM) Policy to the Indias in rural areas as a way of providing quality and affordable health care, low consideration has been given to the system of medicine in rural areas (Kaveri 47). For example, modern medical training, an initiative that is funded by local people has failed to meet the needs of rural dwellers that form the biggest percentage of Indian residents. This is based on lack of proper skills by practitioners to provide adequate health care. For example, 79 % of the practitioners in rural areas who practice allopathic medicine have no appropriate training (Ashok et al 24). Poor accessibility of health care One of the major causes of problem when dealing with health care in Tamil Nadu and other regions is lack of adequate access to health care (Ashok et al 13).This is based on the fact that most of the health care centers are aimed at benefiting the urban dwellers and the upper class (World Health Organization 25). While the health care in urban areas is been provided by properly equipped dispensaries and hospital that are managed by corporate and other organizations, health services in rural areas especially those related to family planning and immunization are addressed by rural health centers that have inadequate facilities leading to high rate of child. Misallocation of financial resources and inadequate public expenditure on health Only 0.9% of the gross domestic product (GDP) is allocated to public health which is not adequate to meet the health needs of rural people Considering that more than 80% of the Indians reside in rural areas, and only 10% of the health budget is directed to rural areas, it is clear that the health problems are real challenges in rural areas (World Health Organization 25). Commercialization of health services Due to the failure of the government to provide adequate drugs in rural hospitals, rural resid ents have continued to seek the services of the private sector. This is despite the contributions of the public towards national budget through income and value added taxes. Based on the high level of poverty by rural residents, it has become a challenge to acquire drugs from the private health care providers. In the same way, drugs that are not recommended by World Health Organization (WHO) have dominated Indian market as dealers focus at maximizing their revenue (World Health Organization 17). Important advice to solving these problems In order to ensure the health of rural people residing in Tamil Nadu and other states is enhanced, it is vital for the government to provide cheap cell phones that are connected to the internet. In this way, communication between health providers will be improved. In addition, it would help in bringing about accessibility of the hospitals by the rural residents . Another way of enhancing accessibility of health care is incresing the number of mobile vans that are already been in use in India. Such vans will also be essential in entering areas where public health services have not reached. It is also important to ensure that states appropriately uses the budgetary allocation of the finacial resources allocated by NRHM. For example, in 2008-09 fiscal year, out of 33.5% of the resources allocated to Uttar Pradesh, 41.7% was unspent (Kaveri, 36). The lack of spending of the

Advanced financial accounting Assignment Example | Topics and Well Written Essays - 1500 words

Advanced financial accounting - Assignment Example Positive Accounting Theory (PAT) involves predictions of choices of firms as pertains accounting policies and the response of firms to any new accounting policies. It also seeks to explain such decision-making actions by the management of different companies. Positive Accounting Theory makes use of theories to draw predictions on the choices management would likely make when selecting accounting policies to implement or use (Deegan, 2009, p.53). According to the theory, the conduct of any firm is in such a way that would maximize its best interests. In this regard, managers would likely do what they feel is best for the company at the expense of the interests of shareholders. In arriving at the choices to pursue, firms are guided by factors within the industry in which they operate. It is such factors on which the positive accounting theory lays a focus. Positive Accounting Theory’s focus is on the relationship that exists between different stakeholders in a business. The stakeholders provide resources to the firm in different capacities. Apart from this relationship, the theory also looks at how accounting would affect the functionality of such relationships. Through agency theory, positive accounting theory explains the possible motivations that guide managers in their choice of preferred accounting methods. In this light, the assumption is that managers, who are agents, would seemingly engage in activities that would create benefits for them at the expense of their principals (Deegan, 2009, p.54). The introduction of restrictive contracts, therefore, comes handy. However, managers still need some freedom to make decisions dependent on the situation. The positive accounting theory has two perspectives namely the efficiency perspective and the opportunistic perspective. The perspectives explain the conduct of managers in as much as choosing accounting policies is concerned. Under the efficiency perspective,

The Blood Clotting Cascade Biology Essay

The Blood Clotting Cascade Biology Essay von Willebrand Factor is a large multimeric clotting protein which plays a significant role in the process of blood coagulation. It is mainly secreted by the vascular endothelial cells and also by megakaryocytes in the bone marrow. The vWF performs two important functions in the process of blood coagulation, and thats why it is very important. First, it is responsible in bringing together the elements to form the primary hemostatic plug. It serves as an anchor for platelets at the site of injury in the blood vessel. Second, it acts as a protective chaperone for Factor VIII, to avoid lysis by proteolytic agents in the blood. The Factor VIII also released by vWF at the site of injury, whereby it brings about the completion of the Intrinsic Pathway of blood coagulation, and seals the site of injury with Fibrin. von Willebrand Disease (vWD) is the most common inherited bleeding disorder in human beings the world over. Although, mutations in the vWF gene are responsible for the type of vWD in a patient, the transmission of vWD to the next generation is not solely linked to the vWF gene, but involves linkages with other genes such as the ABO blood type genes. The gene that encodes von Willebrand Factor is present on the short chromatid of chromosome 12, and is 178kb long with 52 exons or coding sites. Most of the exons are small, some as small as 41 base pairs (Schneppenheim, 2011). Exon 28, which has 1379 base pairs, is the largest. Mutations in the genes encoding vWF are primarily responsible for most vWD cases. Mutations can cause qualitative or quantitative deficiencies of vWF. Mutations in the vWF gene and the vWD that is attributed to the mutations are shown in Figure 1. C:UsersTj WorkDropboxTjBlood SensorThesisvWF Domains rot.jpg Figure : Upper panel: vWF Exons coding for the domains; Lower panel: Locations of mutations and their corrseponding vWD types The multimeric von Willebrand Factor contains identical subunits of 250kDa each. These subunits dimerize (into 500 kilo Daltons subunits) and then multimerize into clusters greater than 10 mega Daltons in weight (Sadler JE, 2006). The vWF performs two functions serve as an anchor for binding platelets to the site of injury and bind to and stabilize Factor VIII from degradation by proteases in the blood and presenting it only at the site of injury. A vWF monomer has a repeated domain structure S D1 D2 D D3 A1 A2 A3 D4 B1 B2 B3 C1 C2 CK (Figure 1). The monomer is 2813 amino acids long. At the N-terminal is the 22 amino acid long signal peptide. Domains D and D3 are specific to Factor VIII binding. Platelets bind to vWF at its A1domain with their Glycoprotein (GP)-1b surface receptors. The A3 domain is specific to collagen, predominantly type III (J. Siekmann, 1998). Thus, domains A1 and A3 are necessary and must be fully functional to form the primary hemostatic plug in the process of coagulation. Figure 2: Domains of vWF protein, (U.S. Department of Health and Human Services, 2007) Overview of the clotting cascade A brief overview of the blood clotting cascade is necessary to understand the function of vWF in the process of clotting. The process of blood coagulation involves platelets and clotting proteins. At the site of injury in a blood vessel, the subendothelial collagen (types I and III) in the extracellular matrix of the blood vessel is exposed to blood. vWF that is present in the blood binds to the exposed collagen with its A3 domain. The flow of blood causes the multimers of the anchored vWF to unfold and expose the sites of platelet binding on the A1 domain (Figure 2). The platelets bind to this domain with their Gp-Ib receptor proteins present on the platelet cell surface. The binding of platelets to vWF activates them and a chemical messenger Thromboxane A2 is released by the platelets. Thromboxane A2 at the site of injury attracts more platelets in the blood, and aids in platelet aggregation. Platelets flowing in the blood stream bind to the activated platelets with a surface prot ein Gp IIb/IIIa. Fibrinogen (Factor I) is present in between the GP-IIb/IIIa receptors of two platelets. Thus, a primary hemostatic plug, though weak in strength, is formed. Figure 3: Sequence of events of blood coagulation due to vWF (U.S. Department of Health and Human Services, 2007) The unfolding of the multimers of vWF also releases Factor VIII at the site of injury. In the Intrinsic pathway of coagulation, Factor VIII is essential in catalyzing the conversion of Factor IX to Factor X, and eventually, prothrombin is catalyzed to from thrombin. Thrombin catalyzes the conversion of Fibrinogen (Factor I) into Fibrin. The fibrin forms a thick proteinaceous mesh, which seals the loss of blood from the blood vessel, this completing the process of hemostasis. Tissue repair and wound healing ensues. von Willebrand Disease von Willebrand Disease (vWD) is a deficiency of von Willebrand Factor. Based on the quantitative and qualitative deficiency, it is classified into subtypes. Combinations of assays are done to detect vWF levels in human plasma. Results of these tests report vWF levels in International Units per deciliter (IU/dL). The plasma concentration of vWF in healthy individuals is reported to be at around 10Â µg/mL (Mannucci, 1998), and the corresponding IU measurement is 100 IU/dL. The classification of the vWD types is based on the criteria developed by the vWF Subcommittee of the International Society of Thrombosis and Haemostasis at Carrboro, North Carolina, USA in 1994. Annual meetings are held by ISTH to review diagnosis and management guidelines for vWD by experts all over the world. The standard guidelines for the diagnosis and treatment of vWD in the USA is based on the vWF Report by the National Heart, Lung and Blood Institute, National Institutes of Health, U.S. Department of Health and Human Services, which was released in 2007 by the expert panel on vWF, chaired by Dr. William L. Nichols, Jr., M.D. The ISTH holds annual meetings all over the world to discuss updates on vWD. The first vWD classification by the ISTH in 1994 was based on information about mutations on the vWF gene. However, because it was appropriate to only a small population of the human race, it was overruled in 2006 and was replaced by the new method based on response to treatment with DDAVP or other blood based therapeutics. vWD is classified based on qualitative and quantitative deficiencies. Partial quantitative deficiency is type 1 vWD and total quantitative deficiency is type 3. Qualitative deficiency is type 2, and is subdivided into types 2A, 2B, 2M and 2N based on the functions of the vWF which are affected. Quantitative deficiencies of vWF are discussed first types 1 and 3 vWD. The main laboratory tests to analyze vWF in patient samples are vWF:Antigen activity (vWF:Ag), Factor VIII: Coagulation activity (FVIII:C) and vWF: Ristocetin Cofactor activity (vWF:RCo). Type 1 vWD A patient with partial quantitative deficiency of vWD is diagnosed as type 1 vWD. The level of vWF in the plasma, though low, can still carry out the formation of the primary hemostatic plug, and also protect Factor VIII. In most type 1 vWD cases, Factor VIII levels are very mildly affected. It is hard to accurately diagnose type 1 vWD because, the vWF levels also depend on the ABO blood grouping. The average vWF level in healthy individuals with blood type O is about 75 IU/dL. It is reasonable to classify the condition of a patient with less than 20 IU/dL vWF level as type 1 vWD because this indicates a probable hereditary mutation. The vWF:Ag and vWF:RCo tests show similar reductions in vWF activity for type 1 vWD patients compared to the reference plasma by ISTH (U.S. Department of Health and Human Services, 2007). Type 3 vWD When the vWF activity of a plasma sample is less than 10 IU/dL, it is classified as type 3 vWD.84-86 Major mutations such as frameshifts, large deletions, splice-site mutations, and missense mutations can be causes for type 3 vWD (U.S. Department of Health and Human Services, 2007). Sometimes, clearance of vWF from the blood stream due to autoimmune disorders can decrease vWF quantity in the blood to type 3 levels of vWD. This is also one of the causes of of Acquired von Willebrand Syndrome AVWS. Type 2 vWD Type 2 vWD is a qualitative deficiency of vWF, where, although the vWF may be produced in normal quantities, fails to perform its tasks effectively. Based on the defect in the von Willebrand Factor, it is mainly classified into types 2A, 2B, 2M and 2N. In type 2A vWD, the vWF platelet binding activity is decreased due to the absence or deficiency of high molecular weight multimers of vWF. There is a sharp fall in the vWF:RCo activity, but not much decrease in vWF:Ag and FVIII:C activity. This is because the vWF is still able to bind to Factor VIII. (Ruggeri ZM, 1980) The high molecular weight multimers are either degraded by proteolytic enzymes in the blood or have not been produced due to mutations in the exons of the vWF gene that code for the A2 and/or the D3 domain. (Schneppenheim R, 2001), (Sutherland JJ, 2004) . Type 2B VWD is characterized by an abnormal increase in the vWF-platelet binding affinity, which leads to depletion of large, functional VWF multimers, and also a fall in platelet numbers (Zimmerman TS, 1986) The platelets circulating in the blood stream are blocked with the mutant vWF, due to which, there is a great difficulty in the formation of the primary hemostatic plug. Thus, thrombocytopenia ensues, along with increased Ristocetin Induced Platelet Aggregation (RIPA) even at low concentrations of Ristocetin. Mutations in the A1 domain are responsible for type 2B vWD (Huizinga EG, 2002). In type 2M vWD the vWF platelet binding activity is reduced. But unlike the type 2A vWD, there is no decrease in the quantities of high molecular weight multimers. This phenomenon is only due to a decreased affinity to the Gp-1b receptors on the platelets (Ginsburg D, 1993), (Schneppenheim R, 2001), (Meyer D, 2001), (Rabinowitz I, 1992) (Mazurier C, 2001). The vWF:Ag, vWF:RCo and FVIII:C activities in types 2A and 2M vWD are similar. They can only be diagnosed based on high resolution gel electrophoresis images. (Meyer D, 2001). Another phenotype of type 2M vWD is the failure of vWF to bind to collagen in the extracellular matrix of the vascular sub-endothelium. vWD type 2N is due to the absence of vWF-Factor VIII binding. This is due to mutations in the D and D3 domains of the vWF protein (Ginsburg D, 1993), (Mazurier C, 2001). The laboratory tests for this type of vWD indicate normal levels for vWF:Ag and vWF:RCo tests, but the FVIII:C activity is only about 10% of normal levels. Acquired von Willebrand Syndrome Acquired von Willebrand Syndrome (AVWS) is type of vWD which is not genetically linked. There are three mechanisms by which it is manifested: Autoimmune reactions against vWF, Increased proteolysis of vWF by a protease ADAMTS13 (A Disintegrin And Metalloproteinase with a ThromboSpondin type 1 motif, member 13), or abnormal increase in the binding affinity of vWF to platelets or other cell surface receptors (U.S. Department of Health and Human Services, 2007). Diagnosis of vWD The diagnosis of von Willebrand Disease and its sub-type is made based on an initial review of previous health conditions and familial history of bleeding disorders, which is done in the clinic, and then obtaining plasma samples of the patient for laboratory tests.

Death in Dickinsons I heard a Fly Buzz When I died Essay -- Emily Dic

Death in Dickinson's I heard a fly buzz when I died Emily Dickinson’s poem â€Å"I heard a fly buzz when I died† is a reflection on what happens when one dies. In the poem, the speaker is waiting to die. It seems as though they are expecting something spectacular to happen at the moment of their death. This spectacular event they are expecting does not happen. I heard a fly buzz when I died By Emily Dickinson I heard a fly buzz when I died; The stillness round my form Was like the stillness in the air Between the heaves of storm. The eyes beside had wrung them dry. And breaths were gathering sure For that last onset, when the king Be witnessed in his power. I willed my keepsakes, signed away What portion of me I Could make assignable,--and then There interposed a fly, With blue, uncertain, stumbling buzz, Between the light and me; And then the windows failed, and then I could not see to see. The speaker describes their situation on their deathbed. The sense of stillness in the air means that the speaker thinks something important is about to happen when they die. Their mood is very solemn, and the still air brings a feeling of tension. There is not yet a mention of the afterlife, just the speaker waiting for death. The stillness is described as â€Å"between the heaves of storm,† referring to the calm that sometimes occurs between storms. This is a very quite almost peaceful stillness, but there is a sign of more things to come. In this case, death is the storm they are waiting for after the calm. The speaker is expecting a big event to happen. As the speakers loved ones begin to mourn, there is a feeling that they too were gathering around to witness some spectacular event that would ... ...There is no large flash of light, no loud noises, just the simple buzzing of a fly. Seeing death as a natural passing places man back in nature. There is no amazing flash of light, or deafening noise, or grand revelation. The choice of a fly as the interrupting entity reinforces the anti-climactic nature of death. Man passes into death the same as anything else in nature. The â€Å"blue, uncertain, stumbling buzz† of the fly implies that not even the buzz of the fly was meant to recognize the death of the speaker. The fly buzzes by because that is the nature of the fly. It did not stop its life for the death of the speaker. This can be seen as nature (the fly) realizing that death is not some incredible event, but a natural passing. Nature moves on, accepting the dead back into the earth. This reinforces that death, even the death of a human, is a simple passing.